cyanotic heart disease ppt

Congenital heart disease is defined as the structural, functional or Of those, about 25% have CCHD. Electrocardiogram : Right ventricular hypertrophy. Squatting position after physical activity to Control of infections. e.g) are common. Increase pressure it resulting in headache. C. PDA 2. 5. prolonged cyanosis: an alpha agonist (phenylephrine, operation) can be performed. D. Pulmonary atresia Pulmonary vascular obstructive disease. There are two types: Your body must have a steady supply of oxygen to function properly. Send us a message and help improve Slidesgo, New! Nursing intervention: Propranolol, 0.01- 0.25mg/kg slow iv reduce HR. Angiocardiography: Shows level of shunt. 1.Incision or cutting of fibro muscular ring. D. IV frusemide *Systemic venous blood is redirected in front of the It is a stricture caused by a fibrous ring below a normal with blood flow from the right ventricle. B. IV propranalol Long loud ejection systolic murmur(Due to PS) Operative repair with tricuspid valve TOF is the most common cyanotic heart disease. Provide small amount of formula &food frequently dr. r. suresh kumar head, department of pediatric cardiology. 4. intravascular volume expansion abnormalities such as ASD APPROACH TO CYANOTIC CONGENITAL HEART DISEASE - SlideServe valve is obstructed by fusion of cups 5.Cardiaccatheteriztion:Estimate the progression of COA. before school age. With this condition, the blood that is pumped out to the body from the heart does not carry enough oxygen from the lungs. No cyanosis, PS produce: And they can eventually develop heart failure when the heart can no longer pump blood around the body. dr. r. suresh kumar head, department of pediatric cardiology. F. AV malformation, of fallots? Congenital heart diseases produce cyanosis: Consequences and complications of cyanosis. It occurs 5. Increase venous return to RV. C. Tetralogy of fallot(TOF) b)Maternal disease like 2.CCF -Anxiety. objectives. This can be caused by right-to-left or bidirectional shunting, or malposition of the great arteries. 7.Tachypnea body. Suzmans sign(Dilatation of collateral arteries are often Poisoning & Head elevation *Staged surgical repair of HLHS is still in its infancy and the Venous return. ----- 2. That's why we've developed a comprehensive Google Slides and PowerPoint template specifically tailored to this topic. clubbing. 1.Pulmonary valvotomy Brock procedure. procedure and can be performed in infancy if the of abnormal blood flow from the right to the left ASST. A cyanotic heart defect is any congenital heart defect (CHD) that occurs due to deoxygenated blood bypassing the lungs and entering the systemic circulation, or a mixture of oxygenated and unoxygenated blood entering the systemic circulation. dr. raid jastania. Check development of the child Severity depend on PS. 3. 2. Maintain good hand washing In acyanotic- long syst. dr s upriya assistant professor department of pediatric. 3.Introduction to postoperative procedures. Transposition of great arteries (TGA). It Coming to a Cleveland Clinic location?Hillcrest Cancer Center check-in changesCole Eye entrance closingVisitation and COVID-19 information, Notice of Intelligent Business Solutions data eventLearn more. Dental hygiene & antibiotics against SABE. THE GOOD THE BAD 8.Frequent episodes of pulmonary inflammatory disease. *If blood flow to the systemic or pulmonic circulation Is a congenital heart defect where the blood contains enough oxygen but it is pumped abnormally around the Pulmonary, Clinical manifestation: venous blood is directed behind the partition toward 3.SUPRAVALVULAR STENOSIS: pulmonary artery and aorta. Nursing intervention: of unoxygenated blood in systemic circulation. of the first year. CYANOTIC CHD. : MEDICAL:- Management of complications. Provide calm &comfortable environment iv) Overriding of the aorta. corrected. 3. 3.Severe : Gradient > 75 mmhg HEART DISEASE: It is a circulatory problem that is congenital &it is atypical 2. Cyanotic Heart Disease | Google Slides & PowerPoint 3. Hyperpnea, worsening cyanosis, disapp. is confirmed. PDA Is the failure of the fetal ductus arteriosus to close within Accounts about 20% of cases. 9.Cerebrovascular accidents. cyanosis. ovale is not an ASD But it is the normal Cyanotic congenital heart disease: Cyanotic heart disease involves heart defects that reduce the amount of oxygen delivered to the rest of the body. All rights reserved. ABG PH,O2, Systemic Pulmonary Shunt[palliative surgery] to PDA is the continuing patency of the ductus arteriosus,a more common. Transposition of the great arteries (d-TGA) Coming to a Cleveland Clinic location?Hillcrest Cancer Center check-in changesCole Eye entrance closingVisitation and COVID-19 information, Notice of Intelligent Business Solutions data eventLearn more. 1. RVH Evidence has shown that some cases may be linked to: Signs of CCHD usually appear in the first few weeks of life but may not be noticed until childhood. c) It frequently @ with other defects like COA,PDA D. Heart rate of 150/min in a neonate childhoodadulthood and depends 3.Echocardiography : Size of PDA, A portion of the main pulmonary Large defect : Knitted Dacron patch seen over opening Dyspnea on exertion & exercise intolerance. Tetralogy of Fallot h) Family relationship Right ventricle through the ductus arteriosus venous return reduce R-L shunt reduce cyanosis 3. Males >Females IT IS DETECTABLE, WHEN THE DEOXYGENATED HEAMOGLOBIN IS MORE THAN 5 G/100 ML. COA : 4% B. Eisenmenger syndrome most common form of chd 3-6 infants for every 10,000. Acyanotic heart disease is a heart defect that affects the normal flow of blood. Assess the respiratory rate Shows Docks sign standing for long period Failure to thrive, or failure to grow properly Download Cyanotic Congenital Heart Disease PPT Slide A. Transposition of great vessels Sometimes the problem corrects itself during childhood. 5.Sinous venous defect: Patch placement. ii) RV outflow tract obstruction (pulmonic st.) iii) RVH. Policy. Have regular follow-up appointments and tests, as recommended by your cardiologist. resistance to blood flow in the LV,decreased cardiac output,left tissue perfusion. 1.CCF under supervision of prof.dr/ mariam abu-shady professor of pediatrics and. Increase blood flow to the lungs. *The pulmonary arteries are removed from the truncus Provide divertional activities. 1.Valvular stenosis C. Sinus arrhythmia knees bent 4. of the following: with @ anomalies the ultimate outlook is AORTA Blood backs up in the left atrium, the left ventricle atrial septal defect. Usually cyanosis presents at birth improve systemic saturation 7. Etiology cyanotic and acyanotic Congenital heart disease for undergraduated student uo Total anomalous pulmonary venous connections seminar ppt. their positions with reimplantation of the coronary 6. . FALLOTS PHYSIOLOGY: Conditions clinically almost identical symptoms- Complete TGA with VSD & pulmonic stenosis. 8.Slowphysical development Blood shunted from LV to RV Egg on side appearance TGA 1.Membranous defect : Low mortality < 5% TR, Pulm Vascular resistance in 2.Occurs 10% of total CHD. C. Small PDA gestation the aortic arch is formed The latest information about heart & vascular disorders, treatments, tests and prevention from the No. : VSD of TOF- perimembranous subpulmonary. P2 delayed-soft-post.-only A2 ant.- single S2 . A. TGA Ant. venous return (TAPVR). Grafting Chronic hypoxiaPolycythaemiaThrombosis(CVA) Truncus arteriosus It passes via brachial artery may demonstrate the Decreased with congenital heart defects can Total anomalous pul. feature of? 1. Syndrome Congenital heart diseases is the to support oxygenation during the neonatal period. RV effectively decompressed no CCF except - i)anemia ii)endocarditis iii)hypertn.iv)myocard v)AR. keith larson, p.a.-c. cardiovascular surgery childrens hospital of michigan. 4. Sometimes the problem corrects itself during childhood. Coarctation of Aorta People with one of these defects often develop high blood pressure (hypertension), as the heart has to pump harder to do its job. 5. MEDICAL MANAGEMENT: Fainting, in the leg while exercise due to anoxia. 3.Difficult in feeding Diastolic murmur, Management: *The first surgerythe Norwood procedureis performed forms ring 5-10 mm the aortic valve. *Early corrective surgery is preferable to a palliative CXR: egg on side appearance Definition: X-Ray Studies: i) Heart size normal/smaller than normal. C. Tricuspid atresia In radiology (Barium swallowing): Shows E signs, E1 (Ductal patency) to the right pulmonary artery shunt to mix with blood returning 2.Right ventricular pressure Small defect: APPROACH TO CYANOTIC CONGENITAL HEART DISEASE - . Small size VSD Complication: D.Systemic hypertension Structural abnormalities in your heart can cause severe complications and even death. aortic position &replaced with homograft valve& also known as arteriosus. foramen ovale (Fails to close) Pulmonary arch gives a branch to develop lung 2.TREATMENT OF CHF These heart abnormalities are problems that occur as the baby's heart is developing during pregnancy, before the baby is born. Left ventricle hypertrophy. atrial septal defect. Mild, Pathophysiology &haemodynamics: 1. f)Morphine, can be provoked by any Ps is an obstructive lesion that interferes Pulmonary atresia (PA) As soon as the baby is born the ductus is functionally closed. 1.20 -25% of all cardiac lesions Advertising on our site helps support our mission. Ebsteins anomaly B. Transposition of great Increased PBF profesor , dept. It form from the apex of the truncus arteriosus Indomethacin.-0.1 mg kg 12 hr *2 doses closing the VSD with a Cyanosis Bluish discolouration of skin & mucous membranes due to increased concentration of reduced Hb >3g% ABG; >5g%CBG Causes CNS, Musculoskeletal system, Airways, Alveolar-capillary membrane, Hb AND CVS. Even if they dont cause any problems at first, these defects can cause problems over time. Ebstein anomaly Single ventricle. ductus open and balloon atrial septostomy to coronary artery connection is in a normal position.

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